Hypotheses on mammalian ageing, toxicity and longevity hormesis - explication by a generalized Gompertz function the role of the "stress protein response" in hormesis DNA repair - as influenced by age, nutrition, and exposure to toxic substances biochemical mechanisms of biphasic dose-responses relationships - role of hormesis effects of low dose radiation on the immune response cellular adaptation as an important response during chemical carcinogenesis biostatistical approaches for modelling u-shaped dose responses curves and study design consideration in assessing the biological effects of two dose doses.
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